Exploring Drug Repurposing in Cancer Treatment

Once you put the patient in this state of nutritional ketosis, it

opens up a whole array, a new array of drugs that may have

been, uh, thrown out, not worked. That's called the whole field of

repurposing

drugs now. But once the body is in this new physiological state,

things that were thought to be ineffective become super

effective. So again, that's a whole new field that will open,

uh, as the results once this information gets

out more.

And you mentioned, um, albendazole in your talk as well.

Does that have a, a gluteamine blocking aspect to it as

well?

Yes, that's in our paper also. That's another interesting thing.

Parasite medications. I don't know about ivermectin because

there was a lot

of political stuff about that. So I didn't-- I said to the, the lab,

"We're not gonna do that," because we didn't wanna get,

get the wrath from somebody. But, but albendazole,

fenbendazole,

um, I said, "Why the hell? Why?" People have taken that and they,

and you hear

people say, "Well, I got rid of my cancer with this

and that."

So I, I, I did a dive on it and I said, "Why, why parasite

medications work against cancer cells?"

And it turns out that the parasites use mitochondrial

substrate-level phosphorylation in the tissue.

And th- and these albendazole, fenbendazole kill these

parasites.

So I tried it on the cancer cell, and sure en- sure as hell, they, it, it

targets the mitochondrial substrate and glycolysis.

So we have a mechanism now why parasite medications are working.

But cancer is not a parasite.

Mm-hmm.

All these people say, "Oh, cancer is a parasite." It's not a parasite.

Parasites and tumors use a common metabolic pathway, and a drug that

works against parasites can be very effective against cancer,

and that's what we

begin to see, especially under nutritional ketosis.

Yeah.

Right? Can you imagine this?

Yeah. Yeah, that-- Oh, sorry. Go ahead.

Um, so ivermectin, it actually works on the mitochondrial

cell death

pathway, the Bcl-2/Bax, Bad pathway.

So it actually helps push the cell death pathway.